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Attendance of the lectures buy cheap zithromax 500 mg online, seminars and practices is compulsory buy generic zithromax 500mg on line, although one may have five absences from the lectures and two absences of in the following distribution: neuroanatomy and neurohistology together: two absences; neurophysiology (seminar and practices) two absences together. If one collects six or more lecture absences (regardless of the reason of the absences) all the exam advantages are withdrawn without further notice. In the case of three or more absences from either the practices or seminars, the verification of the lecture book may be refused. Making up the missed seminars and practices may be possible, but the individual departments determine the actual procedure. If one meets the passing conditions (see below), the end-semester examination may be substituted with the result achieved on the basis of these tests (i. All three departments participate, however, in the second (week 10) and third (week 14) self-controls (both of them are written tests). If someone fails to reach the 60 % in the case of any of the subjects of a department then the student must take the examination on the appropriate subject(s) during the examination period (the actual dates will be determined later). The preconditions of the exam exemption: at least 6 points on histology practicum; at least 12 points on neuroanatomy oral/practicum and at least 12 points on the written tests. Four extra points can be collected from neurophysiology practicals on the 14th academic week during the Neurophysiology closing lab. Nevertheless, the maximum achievable neurophysiology score is 40 points and these extra 4 points are valid only and exclusively in the current academic year (students repeating Neurobiology can register for the end-semester neurophysiology closing lab again. Year, Semester: 2nd year/2nd semester Number of teaching hours: Lecture: 45 Seminar: 15 Practical: 30 1st week: Practical: Study on blood clotting Lecture: Levels of eucariotic gene expression. Practical: Fractionation and quantitative 2nd week: determination of plasma proteins. Significance and interrelationship between 6th week: metabolic, cytokine, hormonal and neuronal Lecture: Tumor suppressor genes and their regulation. Biochemistry of sensitivity of regulation: allosteria, substrate programmed cell death. Signalling Practical: Fractionation and quantitative pathways of nonpenetrating signals. The 7th week: adenylate cyclase and the phospholipase C Lecture: Stress proteins and enzymes in signalling pathway. Heat shock proteins and their The adenylate cyclase and the phospholipase C functions under normal circumstances. Coupling of signaling pathways 9th week: to the regulation of genes and to the actin Lecture: Biochemistry of the blood. Structure and transport, storage and distribution in the human functional domains of fibronectins. Molecular regulation of the iron level in tissue fibronectins, genetic background: cells: stability of transferrin receptor and ferritin alternative splicing. Other adhesion proteins (laminin, Practical: Study on neurotransmitters entactin, thrombospondin, von Willebrand factor, tenascin, etc). Limiting factors , inhibitors and activators of Requirements Requirements for signing the semester: attendance in laboratory practices and seminars. Attendance on the lectures is not compulsory, but recommended: in case of one lecture absence seminar bonus points are erased, in case of two lecture absences all collected points (control test points of the semester) are also erased. Please arrive in time for the lectures, because the door of lecture hall will be closed at the beginning of the lecture. These bonus points will be added to the total points collected during the semester. Every laboratory practices must be performed, if someone is absent due to any serious reason proved by medical papers, the missing experiment have to be performed within the three weeks practice period joining another group (after obtaining permissions from practice teacher of the other group). In case of more than one remedial practice, students cannot get any points for the additional practice units. During the semester 100 (+ 8) points can be collected for the laboratory test (8 points), note book (3 x 4 points) and by the control tests from the material of the lectures (80 points). Bonus points earned by seminar activity will be added to the total points collected during the semester. Semester points will be automatically erased of those students, who break the rules of test writings. Those students who finally reach 70 points in this semester, will get 5 bonus points, those who reach 80 points will get 8 bonus points that will be added to the results of the written part of the exam. Those students, who reaches at least 220 points during the three semesters (Molecular Biology, Biochemistry I. On the written exam 50 points can be collected, it consists of 50 single- and multiple choice test questions from “Molecular Biology” (5 points), “Metabolism” (15 points), “Cell- and organ biochemistry” (25 points) and from the practices of the three semesters (5 points). Oral exam can be taken only if the student collects at least 60 % (30 points) in the written part. The oral part of the examination starts with one basic question of medical orientation, and molecular biology, that should be answered immediately. The list of these questions will be posted on the website at the end of the semester, together with the exam titles of the final exam.
P-62 order 500mg zithromax overnight delivery, P-67 Fisher purchase 500mg zithromax with mastercard, Haran P-1, P-174, P-381 Garcia-Sesma, Alvaro P-261 Eng, Hock-Liew P-497 Fisher, L. O-76 P-181, P-182, P-243, Enne, Marcelo P-42, P-272 Fitzgerald, Mary Grace O-63 P-244, P-257, P-313, P-346 Erbes, Johannes P. P-134 P-359, P-448, P-528 Fondevila, Constantino P-181, P-182, Garrote Lara, Daniel P-22, P-151, P-164 Erez, Danny P-481 P-243, P-244, P-257, P-313 Gasbarrini, Antonio O-73, O-124 Ersoz, Sadik P-424 Fonseca, Eduardo P-30, P-147, Gasloli, Giorgio P-94 Escanhoela, Cecilia A. O-154, P-12 Escobar, Bibiana P-181, P-182 Fontes, Paulo P-122, P-292, P-404 Gasparini, Daniele P-275 Esmailian, Yvonne O-117 Forner, Alejandro P-257 Gastaca, Mikel O-142, P-232, Espinosa, Mª. P-505, P-539 Fouzas, Ioannis P-118, P-344, P-470 Geber, Christian P-280 Eurich, Dennis O-74, P-526 Foxton, Matthew P-293 Gedaly, Roberto O-37, P-314, P-412 Eurotransplant Liver and Intestine Fraga, Enrique O-20 Gelas, Thomas P-51, P-142, Advisory Committee O-152 Franco Gou, Rosa P-395 P-354, P-506, P-525 Eventov- Friedman, Smadar O-91 Francoz, Claire O-26, O-146, Gelbmann, Cornelia M. P-111 Eyraud, Daniel P-119 O-147, P-150 Gelli, Maximilano P-94 Fabregat, Joan O-51, O-142, P-232 Frank, Adam P-235, P-420, P-477 Gentile, Judith W. P-105 Fagiuoli, Stefano O-73, O-124 Franze, Vincent P-2 Gera, Amit P-114 Fahmy, Ahmed O-25, O-48, P-79, P-81 Frauca, Esteban O-4, P-320 Géraldine, Rousseau P-476 Faiz, Omer P-80 Freeman, Richard O-14, O-43, O-49, Gerunda, Giorgio E. P-372 Ghalib, Reem P-224, P-266, P-414, P-463 Fandrich, Fred P-20 Freitas, Maria Cecilia S. O-28 Ghinolﬁ, Davide P-363 Farmer, Douglas O-117, O-155, P-297 Frigo, Anna Chiara P-258 Ghobrial, R. P-225 Froh, Matthias P-111 P-271, P-311, P-441 Fassina, Ambrogio O-38 Frongillo, F. Eugene P-54 Hannoun, Laurent P-119 Giakoustidis, Dimitrios P-344, P-470 Grigoletto, Francesco P-258 Hansen, Bettina E. P-36 Gringeri, Enrico P-92, P-198, P-262 Harden, Cleo P-378 Gil, Luis P-534 Grodzicki, Mariusz P-287 Harlank, Juliane P-41 Gilroy, Richard O-33, P-291 Grollier, Gilles P-384 Harman, Ali O-136 Gimeno, Alberto P-53, P-208, Grossi, Paolo P-28 Hart, Melanie L. O-156, P-110 Gimeno Calvo, Alberto P-203, P-405 Gruessner, Rainer P-331, P-378 Hassanain, M. O-19, P-484, P-496 Ginsburg, Robert O-139 Gruttadauria, Salvatore P-440 Hassoun, Ziad P-44 Giordano, Chris R. P-179 Gruz, Fernando P-518 Hayashi, Masao P-5 Giostra, Emiliano P-426 Guarrera, James V. P-101, P-352, P-402 Haykal, Nadine P-246, P-271, Gitto, Stefano O-44, P-131, P-499 Guckelberger, Olaf P-97, P-153, P-311, P-441 Giuseppe, Bombardieri P-55 P-308, P-473 Haym, Marina Berenguer P-133 Godbole, Gauri P-507 Guedes, Cassia P-42, P-113, P-272 Haznedaroglu, Selcuk P-424 Godoy, Andre P-30, P-147, P-531 Guenther, Rainer P-345 He, Aiqing P-18 Godoy, Iván P-516 Guerra, Juan F. P-366, P-516 He, Jiang-Juan P-393 Gojevic, Ante P-210 Guerrini, Gian Piero O-71, O-83, P-459 Healey, Patrick J. O-103, O-120, Heaton, Nigel O-46, O-139, Goldman, Michel O-17 P-256, P-411, P-462 P-139, P-209, P-278, P-293, Goldstein, Michael P-61 Guido, Cantisani P. O-2, O-100, Gomez, Manuel O-142 Guillaud, Olivier P-51, P-354, P-426 P-108, P-277 Gomez, Miguel A. P-232 Guimaraes, Teresa P-30 Hebbi, Hammood P-307, P-330, Gómez-Arellano, Graciela C. P-163 Gontarczyk, Gajusz P-303 Gulati, Reema P-322 Heifets, Michael P-18 Gonzales, Ana Carolina P-113 Gülay, Hüseyin P-250 Height, Sue P-139 Gonzalez, Ana Carolina P-42, P-272 Gunasekaran, G. P-90, P-98 Gonzalez, Andres P-386 Gunson, Bridget O-106 Heise, Michael P-112, P-178, Gonzalez, Ignacio P-232 Gupta, Ankit P-46, P-421 P-184, P-449 Gonzalez, Javier P-273, P-415, P-521 Gupta, Subash P-536 Helena, Ernani T. P-251 Heneghan, Michael O-2, O-11, Gonzalo, Sapisochin O-70, P-99 Guy, Stephen O-99 O-100, P-277, P-293 Gopasetty, Mahesh S. P-39 Ha, Tae-Yong O-64, O-129, O-131, Hengstler, Jan P-20 Goralczyk, Armin P-100 P-70, P-165, P-329, P-445 Henry, Scot D. P-54 Haberal, Mehmet O-62, O-133, O-136, Hernandez, Francisco O-4, P-320 Gordon, Sherilyn O-117 P-75, P-137, P-260, Hernandez-Alejandro, Roberto P-401 Gordon Burroughs, Sherilyn P-387 P-299, P-466, P-522 Herrero, Ignacio P-65, P-348 Gordon-Burroughs, Sherilyn P-297 Habib, Tikvah Y. P-90 Habrecht, Olaf O-3, P-112, P-425 Herrine, Steven P-235, P-420 Goss, John P-152, P-271 Hadzic, Dino P-139 Hierro, Loreto O-4, P-320 Goto, Hidemi P-83 Hadzic, Nedim P-509 Hilmi, Ibteasm A. O-57 Goto, Shigeru P-190 Haﬂiger, Silvia P-467 Hindennach, Milo P-249 Goulis, Ioannis P-118, P-344, P-470 Hage, Antoine P-104 Ho, Cheng-Maw Dominic O-35 Gouvêa, Glauber P-374 Hagspiel, Klaus P-464 Ho, Ming-Chih O-35 Gouw, Annette S. P-188 Hajdu, Cristina O-134 Hochhauser, Edith O-85 Gouya, Hervé O-143 Halac, Esteban T. O-141, P-135 Hoekstra, Harm O-45, P-195, P-388 Goyal, Neerav P-536 Halpern, Márcia P-113 Holland, Bart P-428, P-482 Gramenzi, Annagiulia P-131, P-499 Ham, John P-34 Hollinger, Blaine P-224 Grandadam, Stephane P-489 Hamberg, Karin J. O-54 Hamid, Malik P-361 Homayounfar, Kia P-100 Gravenstein, Nik P-179, P-340 Hammoudeh, Saeb P-242 Honarpisheh, Human P-345 Grazi, Gian Luca O-21, O-44, P-131, Hammoudi, Saeb P-214, P-338, P-339 Hong, Jhonny P-297 P-171, P-359, P-448, P-499, P-528 Hamshow, Mohammad M. P-337 Hong, Johnny O-155, P-385, P-474 Gregorio, Germana P-439 Hamza, Alaa P-317, P-315 Honoré, Pierre P-206 Greig, Paul O-50, O-65, P-77, P-288 Hamza, Alla F. P-200 Han, Young Seok P-211 Hoshino, Ken O-5, P-16, P-508 Gridelli, Bruno P-169, P-335, P-440 Hanish, S. P-241 S286 Author Index Hrehoret, Doina P-154 Ismail, Mohammad O-123 Juárez-Uriarte, Cuauhtemoc P-375 Hrstic, Irena P-106, P-210 Isola, Miriam P-123 Jude, Brigitte P-376 Hsieh, Chung-Bao P-216, P-495 Ito, Shuichi O-110 Julià, David O-51 Hsu, Li-Wen P-190 Ito, T. P-433 Jun, In Gu O-58 Hu, Ke-Qin P-385 Itxarone, Bilbao O-70, P-99 Juneja, Rajiv P-295 Hu, Rey-Heng O-35 Iyer, Kishore P-363 Jung, Dong Hwan P-165, P-329 Hua, Ran Jiang P-397 Jabbour, Nicolas O-47 Jung, DongHwan P-69 Huang, Chia-Jung P-4, P-377 Jabiry-Zieniewicz, Zoulikha P-490, P-493 Jung, Dong-Hwan O-64, P-70, P-445 Huang, Li-Tung P-196 Jacob, Dietmar A. P-500 Jacquelinet, Christian O-30 Jung, Jae Pil P-541 Huang, Shiu-Feng Kathy O-35 Jah, Asif P-514 Jureczko, Lidia P-175 Huang, Tung-Liang P-326, P-327, P-328 Jain, Ashok P-519 Juricic, Danica P-210 Huang, Yi P-267 Jain, Ashokkumar O-18, P-80, Kabamba, Benoît P-44 Hudson, Mark P-480 P-115, P-173 Kadry, Zakiyah P-26 Hughes, Michael G. P-11, P-229, Jakoby, Estrella P-45, P-49, P-168 Kafshi, Arash P-77, P-288 P-231, P-296, P-486 Jalil, Sajid P-267 Kahn, Khalid P-378 Hughes, Michel P-39 Jamieson, N.
Associated symptoms may include breath- pnoea an underlying cause should be sought zithromax 100 mg low cost, such as lessness purchase zithromax 250 mg without prescription, dizziness, syncope and/or chest pain. This symptom normally arises when a patient’s exer- r Palpitations lasting just a few seconds are often due cise tolerance is already reduced. The patient becomes aware of the mechanisms are responsible for this phenomenon: a pause that occurs in the normal rhythm after a prema- redistribution of ﬂuid through gravity in the lungs ture beat and may sense the following stronger beat. Some patients may know how to terminate propping themselves up on pillows at night, or, in se- their rapid palpitations with manoeuvres such as vere cases, sleeping in a chair. Orthopnoea is highly squatting, straining or splashing ice-cold water on the suggestive of a cardiac cause of dyspnoea, although it face. These features are very suggestive of a distinct may also occur in severe respiratory disease due to the tachyarrhythmia rather than general anxiety or pre- second mechanism. It is thought to occur by a simi- Syncope lar mechanism to orthopnoea coupled to a decreased sensory response whilst asleep. Patients awake breath- Syncope is deﬁned as a transient loss of conscious- less and anxious, they often describe having to sit up ness due to inadequate cerebral blood ﬂow. Cerebral Chapter 2: Clinical 25 perfusion is dependent on the heart rate, the arterial cases the pain causes the patient to limp, hence the term blood pressure as well as the resistance of the whole vas- claudication and the pain characteristically disappears culature. There may be no warning, or patients may describe feel- The distance a patient can usually walk on the ﬂat be- ing faint, cold and clammy prior to the onset. Asthenarrowing tend to be ﬂushed and sweaty but not confused (unless ofthearteriesbecomesmoresigniﬁcant,theclaudication prolonged hypoxia leads to a tonic-clonic seizure). Eventually rest pain may occur, this r Vasovagal syncope is very common and occurs in the often precedes ischaemia and gangrene of the affected absence of cardiac pathology. The heart contracts force- fully, which may lead to a reﬂex bradycardia via vagal Oedema stimulation and hence a loss of consciousness. A number of mechanisms tion, hypovolaemia or due to certain drugs especially arethoughttobeinvolvedinthedevelopmentofoedema. Normally tissue ﬂuid is formed by a balance of hydro- r Cardiac arrhythmias may result in syncope if there is a static and osmotic pressure. This may oc- Hydrostatic pressure is the pressure within the blood cur in bradycardias or tachycardias (inadequate ven- vessel (high in arteries, low in veins). The loss of consciousness occurs produced by the large molecules within the blood (albu- irrespective of the patient’s posture. A Stokes–Adams min, haemoglobin) and draws water osmotically back attack is a loss of consciousness related to a sudden into the vessel. The hydrostatic pressure is high at the loss of ventricular contraction particularly seen dur- arterial end of a capillary bed hence ﬂuid is forced out of ing the progression from second to third degree heart the vasculature (see Fig. The colloid osmotic pressure then draws ﬂuid back in r Carotid sinus syncope is a rare condition mainly seen at the venous end of the capillary bed as the hydrostatic in the elderly. As a result of hypersensitivity of the carotid sinus, light pressure, such as that exerted by atight collar, causes a severe reﬂex bradycardia and hence syncope. The syncope results from an inability of the heart to increase cardiac output in response to in- Hydrostatic Oncotic 0ncotic Hydrostatic creased demand. Intermittent claudication Artery Vein Claudication describes a cramp-like pain felt in one or both calves, thighs or buttocks on exertion. This may be a result of blood bypassing ﬂuid is then returned to the circulation via the lymphatic the lungs (right to left shunting) or due to severe lung system. Mechanismsofcardiovascularoedemaincludethefol- lowing: r The arterial pulse Raised venous pressure raising the hydrostatic pres- sure at the venous end of the capillary bed (right ven- The pulse should be palpated at the radial and carotid tricularfailure,pericardialconstriction,venacavalob- artery looking for the following features: struction). The normal pulse is deﬁned as a rate be- which increases the circulating blood volume with tween 60 and 100 beats per minute. Outside this range pooling on the venous side again raising the hydro- it is described as either a bradycardia or a tachycardia. Albumin is the major factor respon- r The character and volume of the pulse are normally sible for the generation of the colloid osmotic pressure assessedatthebrachialorcarotidartery. A drop volume felt at the carotid may be described according in albumin therefore results in an accumulation of to the waveform palpated (see Fig. Radio-femoral delay is suggestive of coarcta- is left after pressing with a thumb for several seconds) tion of the aorta, the lesion being just distal to the or nonpitting. Cardiac oedema is pitting unless long origin of the subclavian artery (at the point where the standing when secondary changes in the lymphatics may ductus arteriosus joined the aorta). Distribution is dependent lay suggests arterial occlusion due to an aneurysm or on the patient. Pleural effusions and Jugular venous pressure ascites may develop in severe failure. The internal jugular vein is most easily seen with the pa- tient reclining (usually at 45˚), with the head supported Cyanosis and the neck muscles relaxed and in good lighting con- Cyanosis is a blue discolouration of the skin and mu- ditions. It is due to the presence of desaturated toid muscle in the upper third of the neck, behind it haemoglobin and becomes visible when levels rise above in the middle third and between the two heads of ster- 5 g/dL. Cyanosis is not present in very anaemic patients nocleidomastoid in the lower third. Cyanosis is divided from the carotid pulse by its double waveform, it is non- into two categories: palpable, it is occluded by pressure and pressure on the r Peripheral cyanosis, which is seen in the ﬁngertips and liver causes a rise in the level of the pulsation (hepato- peripheries.